Category: T-Type Calcium Channels

In addition, ApE3-42 and A4-42 when expressed together elicited neurological deficits, which were significantly stronger as compared to expression of ApE3-42 or A4-42 alone [43]

By spierarchitectur

March 5, 2023 Off

In addition, ApE3-42 and A4-42 when expressed together elicited neurological deficits, which were significantly stronger as compared to expression of…

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By spierarchitectur

December 16, 2022 Off

M. gland, the main source of adrenaline under nerve-racking situations.Prez-Alvarez, A., Hernndez-Vivanco, A., McIntosh, J. M., Albillos, A. Native 64*…

Moreover, ERK2 is not involved in the S118 phosphorylation that takes place following the hER N-terminal region ligand-dependent association with the C-terminal part of the protein (Mtivier phosphorylation assays (Figures?4 and ?and5)

By spierarchitectur

April 9, 2022 Off

Moreover, ERK2 is not involved in the S118 phosphorylation that takes place following the hER N-terminal region ligand-dependent association with…

In summary, our results suggest that compounds from might be promising candidates for molecular target-based cancer prevention and adjuvant therapy

By spierarchitectur

February 6, 2022 Off

In summary, our results suggest that compounds from might be promising candidates for molecular target-based cancer prevention and adjuvant therapy.…

﻿In addition, ApE3-42 and A4-42 when expressed together elicited neurological deficits, which were significantly stronger as compared to expression of ApE3-42 or A4-42 alone [43]

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﻿Moreover, ERK2 is not involved in the S118 phosphorylation that takes place following the hER N-terminal region ligand-dependent association with the C-terminal part of the protein (Mtivier phosphorylation assays (Figures?4 and ?and5)

﻿In summary, our results suggest that compounds from might be promising candidates for molecular target-based cancer prevention and adjuvant therapy

In addition, ApE3-42 and A4-42 when expressed together elicited neurological deficits, which were significantly stronger as compared to expression of ApE3-42 or A4-42 alone [43]

M

Moreover, ERK2 is not involved in the S118 phosphorylation that takes place following the hER N-terminal region ligand-dependent association with the C-terminal part of the protein (Mtivier phosphorylation assays (Figures?4 and ?and5)

In summary, our results suggest that compounds from might be promising candidates for molecular target-based cancer prevention and adjuvant therapy